Kwayar cutar, wadda ta rage murfin hagu, kuma a cikin mafi yawan lokuta da ke da hankali na zuciya, ana kira hypertrophic cardiomyopathy (HCMC). A cikin wannan cututtuka, ɗaukar matsala a lokuta da yawa na faruwa a hankali, sabili da haka ne sau da yawa ana iya lalata septum.
An yi imani da cewa wannan wata cuta ce ga 'yan wasa - saboda karuwar karfin jiki da ake yi wa hypertrophy. Mun riga mun san lokuta da dama yayin da 'yan wasan suka mutu akan wasanni saboda wasan motsa jiki - dan wasan kwallon kafa na Hungary Miklos Feher da dan wasan Amurka Jesse Marunde.
A cikin wannan cuta, ƙwayoyin tsoka a cikin myocardium suna da matsananciyar wuri, wadda ke hade da maye gurbi.
Nau'i na hypertrophic cardiomyopathy
A yau, likitoci sun gano 3 nau'o'in hypertrophic cardiomyopathy:
- Ƙaramar Basal - mai hankali a hutawa ya fi ko kuma daidai da 30 mm Hg. Art.
- Haɗin Labile - haɗuwa marar kyau na haɓakaccen ƙwararru na intraventricular ana kiyaye su.
- Latent obstruction - wani gradient a cikin ƙasa mai kwantar da hankali kasa da 30 mm Hg. Art.
Hanyoyin cututtuka na kyakken jini sun dace da waɗannan nau'i uku na cutar, yayin da ainihin ba shi da haɓakawa yana nuna wani ƙwararren stenosis na kasa da 30 mm Hg. Art. a cikin kwanciyar hankali da fushi.
Bayyanar cututtuka na hypertrophic cardiomyopathy
Bambanci na hypertrophic cardiomyopathy na iya zama babu - kimanin kashi 30 cikin marasa lafiya ba su yin komai ba, wanda idan aka yi mutuwa ba zato ba ne kawai bayyanar cutar. A wani yanki na musamman akwai marasa lafiya wadanda basu kula da kukan ba, sai dai saboda damuwa na zuciya.
Domin wannan cuta tana da alamar ƙwayar ƙananan ƙwayar cuta - a cikin wannan yanayin, rashin lalacewa, rashin ƙarfi da numfashi, kuma hare-haren angina ya faru.
Bugu da ƙari, tare da cututtukan zuciya na jini, akwai alamun rashin cin nasara na zuciya na ventricular hagu, wanda zai iya haifar da mummunan zuciya.
Rashin ƙaddara a cikin ɓacin zuciya zai iya haifar da ɓarna . Sau da yawa waɗannan su ne ƙananan extrasystoles da ventricular tachycardia.
A cikin lokuta masu yawa, marasa lafiya na iya samun ciwon cututtuka da kuma thromboembolism.
Sanin asali na hypertrophic cardiomyopathy
Sabanin sauran nau'o'in cardiomyopathy, ana iya bincikar samfurin hypertrophic mafi sauki saboda ka'idar da aka kafa: don ganewar asali da za a yarda da shi, katakon na katsewa ya kamata ya fi girma ko kuma daidai da 1.5 cm tare da kasancewar dasfuncin ventricular hagu (rashin hutu).
Lokacin da aka bincika, ana samun mai haƙuri don fadada iyakar zuciya zuwa hagu, kuma lokacin da aka katse, an ji motsin (systolic rhomboid).
Daga cikin ƙarin hanyoyin nazarin wannan alamun sune:
- electrocardiography - tare da shi sun gano wani karuwa a hannun hagu atrium;
- Rubutun kalmomi shine hanya mai mahimmanci wanda ke taimakawa wajen gano ma'anar hypertrophy;
- Kulawa ECG - ƙayyade matsalar rikice-rikice;
- Halin yanayin halayen magnetic - amfani da shi azaman hanyar tsaftacewa;
- ilimin kwayoyin halitta shine daya daga cikin hanyoyin da za'a fi dacewa don tantance cutar.
Jiyya na hypertrophic cardiomyopathy
Sanin asali da kuma maganin cututtukan hypertrophic cardiomyopathy suna da alaka da juna don hana mummunan sakamako. Bayan an kimanta mahimmancin alamun cutar, idan akwai yiwuwar wani sakamako na mutuwa, ana yin magani mai mahimmanci. Idan babu barazanar mutuwa, kuma bayyanar cututtuka ba
Don magani yana da mahimmanci don rage yawan aikin jiki, da kuma yin amfani da kwayoyi tare da sakamako mai tasiri na ionotropic. Wannan rukuni ya hada da beta-blockers da mastacin antagonists. An zaba su a kowanne ɗayan, kuma an ba da wannan liyafa na dogon lokaci (har zuwa gadon liyafar rayuwa), likitoci a yau suna ƙoƙari su tsara magunguna tare da ƙananan sakamako masu illa. Ana amfani da tsohon Anaprilin, kuma a yau akwai wasu analogues na sabon ƙarni.
Har ila yau, ana amfani da magunguna da maganin rigakafin maganin maganin maganin maganin cututtuka.